Hepatitis C virus (HCV) gains entry into susceptible cells by interacting with cell surface receptor(s). Viral entry is an attractive target for antiviral development because of the highly conserved mechanism.

Authors: Takuya Matsumura, Zongyi Hu, Takanobu Kato, Marlene Dreux, Yong-Yuan Zhang, Michio Imamura, Nobuhiko Hiraga, Jean-Marc Juteau, Francois-Loic Cosset, Kazuaki Chayama, Andrew Vaillant, T Jake Liang

Read publication

Back to Resources